Tswvyim 1: Hyperuricemia yog gout
Coob leej neeg ntseeg tias muaj uric acid nyob rau hauv cov ntshav siab yog gout thiab yuav tsum tau kho. Qhov tseeb, txawm hais tias hyperuricemia yog qhov tseem ceeb tshaj plaws biochemical hauv paus ntawm gout, nws tsis yog lub ntsiab lus rau gout. Gout yog ib yam kab mob uas tshwm sim los ntawm qhov txawv txav purine metabolism thiab / lossis txo qis uric acid excretion. Cov yam ntxwv kho mob yog hyperuricemia, rov tshwm sim mob monoarthritis, gout pob zeb deposition tshwm sim los ntawm sodium urate, thiab mob gout pob zeb mob caj dab. Yog tsis muaj kev kho mob, lawv feem ntau tsim mus rau gouty nephropathy.
Tswvyim 2: Kev txwv tsis pub noj zaub mov muaj purine txaus txaus
Cov kev tshawb fawb tau taw qhia tias txawm tias kev siv cov khoom noj uas tsis tshua muaj purine cov ntsiab lus yog nruj me ntsis txwv, qhov txo qis hauv cov ntshav uric acid concentration yog txwv; Ntawm qhov tsis sib xws, yog tias tsis muaj kev noj zaub mov kom zoo, nws tuaj yeem ua rau cov ntshav uric acid nce nrawm mus rau lub xeev uas nws tuaj yeem tawm tsam txhua lub sijhawm. Ntawm qhov tod tes, cov neeg mob gout feem ntau nrog hyperlipidemia, kub siab lossis ntshav qab zib. Cov kab mob no xav tau kev tswj hwm zaub mov, yog li kev tswj hwm kev noj haus yog tsim nyog.
Gout txoj hauv kev noj zaub mov yog kom tsis txhob muaj cov khoom noj purine siab, xws li tsiaj viscera, sardine, clams, crabs, thiab lwm yam. Muaj cov purines nruab nrab xws li ntses thiab cws, nqaij, peas, spinach, thiab lwm yam. Raws li cov txiv hmab txiv ntoo, zaub, mis nyuj, qe, thiab lwm yam, cov ntsiab lus ntawm purine kuj tsawg.
Tswvyim 3: Kev txom nyem los ntawm gout tshwm sim los ntawm kev noj zaub mov tsis raug
Muaj ob pawg tseem ceeb ntawm cov neeg uas muaj mob gout: cov txiv neej hnub nyoog 40 txog 60 xyoo, thiab cov neeg uas haus cov zaub mov muaj roj ntau thiab cawv. Cov poj niam feem ntau yog postmenopausal, tshwj xeeb tshaj yog cov neeg uas muaj ntshav siab thiab noj diuretics.
2. Kev rog rog nrog hyperuricemia, hypertriglyceridemia, hyperglycemia, thiab kub siab.
Noj cov tshuaj uas cuam tshuam rau uric acid excretion, xws li diuretics thiab tshuaj aspirin tsawg.
4. Muaj tsev neeg keeb kwm ntawm gout, los yog taug qab keeb kwm ntawm cov pob zeb tso zis hauv cov neeg txheeb ze.
Tswvyim 4: Qhov chaw ntawm gout tawm tsam yog txwv rau pob qij txha
Hauv 70 feem pua ntawm cov neeg mob gout, thawj qhov chaw pib yog nyob rau hauv thawj tus ntiv taw loj (thawj metatarsophalangeal sib koom). Kev mob kuj tuaj yeem tshwm sim hauv dorsum ntawm ko taw, Achilles leeg, pob taws, pob taws, thiab hauv caug pob qij txha. Tsis tas li ntawd, cov pob qij txha sab sauv xws li ntiv tes thiab lub luj tshib kuj tuaj yeem tshwm sim. Ntau tshaj 90 feem pua ntawm cov neeg mob muaj lawv thawj qhov pib ntawm cov pob qij txha qis.
Vim li cas cov pob qij txha qis, tshwj xeeb tshaj yog thawj tus ntiv taw loj, ua rau qaug dab peg? Qhov laj thawj tseem tsis tau paub meej. Txawm li cas los xij, kev tawm tsam gout tshwm sim tom qab uric acid dhau los ua sodium urate crystals, thiab o tshwm sim. Yog li ntawd, gout tuaj yeem tshwm sim nyob rau hauv qhov chaw uas muaj crystallization. Crystallization yog nquag tshwm sim nyob rau hauv cov cheeb tsam uas muaj zog acidity, nquag txav, load-bearing cheeb tsam, thiab tsis kub. Yog li ntawd, cov cheeb tsam uas crystallization yuav tshwm sim yog pob qij txha, auricles, ob lub raum, urinary ib ntsuj av, thiab lwm yam. Cov chaw no yog raws li nyob rau hauv ntau yam mob uas yuav ua rau crystallization, thiab pob qij txha, tshwj xeeb tshaj yog thawj tus ntiv taw loj, raws li tag nrho cov mob.
Tswvyim 5: Kev kho mob tuaj yeem nres tom qab uric acid ruaj khov thiab qhov mob tau txo qis
Kev kho mob ntawm gout yuav tsum ua raws li kev noj haus thiab kev noj zaub mov kom zoo. Qee cov neeg mob tso tseg cov tshuaj tom qab lawv cov uric acid stabilized. Txawm li cas los xij, nrog xws li kev tshem tawm tsis tu ncua, uric acid sai sai tau nce ntxiv. Txoj kev kho kom raug yog tso cai rau tus kws kho mob maj mam txo cov koob tshuaj tom qab uric acid poob rau lub hom phiaj tus nqi (cov ntshav uric acid 300-360 umol / l). Ua ntej ntawd, nws yog ib qho tsim nyog yuav tsum tau ua raws li cov tshuaj tsis tu ncua.
Feem ntau, kev kho mob gout tau muab faib ua peb theem, raws li hauv qab no:
Theem 1 yog kev kho mob gout. Qhov no yog kev kho mob rau qhov mob nyem, feem ntau nrog cov tshuaj tiv thaiv kab mob thiab tshuaj analgesic.
Qhov thib ob theem yog thawj zaug kev kho mob ntawm kev tswj uric acid tom qab lub gout nres ploj. Lub sijhawm siv tshuaj, ua ke nrog kev noj haus thiab kev saib xyuas txhua hnub, yog 3-6 hli. Hom tshuaj siv sib txawv nyob ntawm seb hom hyperuricemia. Nyob rau hauv txoj cai, cov neeg mob uas tsis tshua muaj excretion yuav tsum siv uric acid excretion txhawb cov tshuaj, thaum cov neeg mob uas muaj ntau ntau lawm yuav tsum siv cov tshuaj uas inhibit uric acid synthesis.
Theem 3 yog ua raws li kev kho mob thawj zaug rau kev tswj uric acid, nrog rau kev kho mob uric acid nyob rau lub neej, nrog rau kev noj tshuaj thiab kev noj haus kom tsis txhob muaj teeb meem lossis rov tshwm sim dua.
